2 edition of Study of some factors and a possible mechanism of action involved on gastric ulcers (gizzard erosion) in chicks found in the catalog.
Study of some factors and a possible mechanism of action involved on gastric ulcers (gizzard erosion) in chicks
Emilio Castro Campos
Written in English
|Statement||by Emilio Castro Campos.|
|The Physical Object|
|Pagination||ix, 55 leaves ;|
|Number of Pages||55|
Abstract. Background. Paeonia extract mixture HT is a standardized multiherbal mixture comprising extracts from Inula britannica flowers and Paeonia lactiflora roots, which are used to treat digestive disorders in traditional Korean medicine. This study was focused on elucidating the underlying mechanisms of the gastroprotective effects of HT in different gastric ulcer models. In addition, cigarette smoking interferes with prostaglandin synthesis, which compromises defense mechanisms. A genetic predisposition seems to exist for the development of PUD. Infection by H. pylori is thought to be associated with as many as 90% of duodenal and 70% of gastric ulcers. Chronic emotional stress was previously believed to cause.
The present study investigated whether isopulegol, a monoterpene present in essential oils of several aromatic plants, would be able to promote some gastroprotective effect and also verified the possible mechanisms involved in this action. For this study, ethanol- and indomethacin-induced gastric ulcer models in mice and histopathological. The mechanisms involved in the cold stress-induced ulceration are: disturbance in gastric secretion, alteration in microcirculation of gastric mucosa and abnormal gastric motility (Kitagawa et al., ). Central mechanism including vagal over activation has also been considered for pathogenesis of stress ulcers.
traditionally used to relieve pain from gastric ulcers. Objective: This study was designed to determine the antiulcer activity of a methanol extract of M. calabura leaves (MEMC) and the possible mechanisms of action involved. Materials and methods: An acute toxicity study was conducted using a single oral dose of mg/kg MEMC. Clinical signs associated with gastric ulcers are not specific and can always potentially be explained by other non-gastric factors. Thus far, investigation of potential diagnosis via blood or urine sampling has not proved useful (for example, O’Conner et al, ) and, therefore, diagnosis depends entirely on .
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Background. Gastric ulcer is a disease that affects many people around the world, and its progression is attributed to an imbalance between aggressive factors (acid, pepsin, Helicobacter pylori, stress, alcohol, and continued use of non-steroidal anti-inflammatory drugs), and protective factors (mucus, bicarbonate, prostaglandin, blood flow, the antioxidant system, sulfhydryl compounds, nitric Cited by: 8.
Some authors refer to gastric ulcers as the new “plague” of the 21st century. It has been projected that million of the worldwide population are affected by gastric ulcers with a mortality rate of million. The pathophysiology of gastric ulcer is associated with the imbalance between aggressive and protective factors in the by: Gastric and duodenal ulcers are breaks in the gastric and duodenal mucosa.
Both gastric and duodenal ulcers relate to the corrosive action of pepsin and hydrochloric acid on the mucosa of the upper gastrointestinal tract. Ulcers generally range between. In the present study, we investigated whether violacein can promote a gastroprotective effect and verified the possible mechanisms involved in this action.
For this study, an indomethacin-induced. In this model, the gastric lesions are induced by the inhibition of prostaglandin synthesis. Male Swiss mice were randomly divided into four groups, treated orally with vehicle (controls), hecogenin (15, 30, 60 and 90 mg/kg, p.o.) and ranitidine ( mg/kg, p.o.) as reference.
After 60 min, the gastric ulcers were induced in all groups by indomethacin (20 mg/kg, p.o.) suspended in % Cited by: Since ulcerogenic factors such as Helicobacter pylori, non-steroidal anti-inflammatory drugs and stress can increase expression of inflammatory cytokines in gastric mucosa, gastric mucosal inflammation may play key roles in ulcer recurrence.
In acetic acid-induced gastric ulcers, persistent infiltration of neutrophils into scarred mucosa, which is caused by prostaglandin deficiency, affects future ulcer. the%absorption%of%some%nutrients%and%other%medications.%Omeprazole%depletes% Vitamin%Bb12%stores,%causing%acobalamin%deficiency.%The%only%pertinentside%.
Several mechanisms of action have been described to explain the development of bowel perforation as a result of bevacizumab. In the first mechanism, the inhibition of VEGF by bevacizumab could cause thrombosis of smaller splanchnic or mesenteric vessels, leading to bowel ischemia and ultimately bowel perforation.
5,6 Vascular endothelial growth factor is involved in cytoprotection. The nurse is discussing the mechanism of action of omeprazole (Prilosec) with a group of nursing students. Which statement by a student would best indicate understanding of the mechanism of action of omeprazole.
"Absorption occurs in the stomach when the pH is greater than 5." b. "It irreversibly inhibits the enzyme needed to produce stomach. A STUDY OF THE FACTORS INVOLVED IN THE PRODUCTION OF GASTRIC ULCERS BY THE RESTRAINT TECHNIQUE DAVID A. BRODIE, PH.D., AND HARLEY M.
HANSON, PH.D. From the Merck Institute for Therapeutic Research, West Point, Pennsylvania It is well known that certain situations are capable of inducing gastrointestinal ulcers in both man and experimental ani mals. A peptic ulcer is a sore on the lining of the stomach or duodenum. The two most common types of peptic ulcer are called “gastric ulcers” and “duodenal ulcers”.
Smoking, another possible confounding factor, has been linked to duodenal ulcers, 28,29 gastric ulcers, 29 and gastric cancer. 30 In our study, we found an increased risk of virtually all types of. Ellagic acid, a compound of proven antiulcer activity, was found in MELP as the major component.
These results confirmed the traditional use of MELP in Brazilian popular medicine for the treatment of gastric ulcers and shed some light on some of its mechanisms of action.
Peptic ulcer disease (PUD) is a break in the inner lining of the stomach, the first part of the small intestine, or sometimes the lower esophagus. An ulcer in the stomach is called a gastric ulcer, while one in the first part of the intestines is a duodenal ulcer. The most common symptoms of a duodenal ulcer are waking at night with upper abdominal pain and upper abdominal pain that improves.
The goals of pharmacotherapy are to eradicate H pylori infection, to reduce morbidity, and to prevent complications in patients with peptic ulcers. Acid suppression is the general pharmacologic principle of medical management of acute bleeding from a peptic ulcer, using histamine-2 receptor antagonists (H2RAs) and proton pump inhibitors (PPIs).
Excessive gastric acid is only one of the potential “hostile” factors in Peptic Ulcer Disease. Decreased mucosal defense is another potential factor.
Some “protective” factors such as prostaglandins, mucus, bicarbonate and blood ﬂow in the mucosa can be disrupted by the “hostile” factors, causing a peptic ulcer. Gastric and duodenal ulcers are both types of peptic ulcers. These ulcers can cause different symptoms, depending on where they are.
A peptic ulcer on the inside of the stomach lining is a gastric. Gastric ulcer is a disease that affects many people around the world, and its progression is attributed to an imbalance between aggressive factors (acid, pepsin, Helicobacter pylori, stress, alcohol, and continued use of non-steroidal anti-inflammatory drugs), and protective factors (mucus, bicarbonate, prostaglandin, blood flow, the antioxidant system, sulfhydryl compounds, nitric oxide and.
- A family history of ulcers. More than 20% of people with peptic ulcers also have a family history of the problem. - Increased age. Studies show that peptic ulcers. In some people, the H pylori bacteria cause an infection in the lining of the stomach, which may lead to gastric ulcers.
Damage to the stomach lining from stomach acid increases the likelihood that H pylori infection will result in a gastric ulcer. Other risk factors for gastric ulcer include alcohol use, tobacco use, and prolonged use of medications such nonsteroidal anti-inflammatory.
Gastric ulcers, also known as stomach ulcers are one of two types of ulcers that affect the stomach and small intestine. An ulcer can be defined as a break in the continuity of the skin or mucous lining due to multiple reasons such as acid overload, drug abuse, stress, and infections.of those with gastric ulcers.4 It is typically The bacterium is a known cause of gastric and duodenal ulcers5 and is a risk factor for mucosa-associated lymphoid tissue (MALT).
Researchers Study Supplement’s Effects on Gastric Ulcers. the more severe the ulcers. The study horses had not received any ulcer treatment for at .